Environmental factors of equine osteochondrosis and fetlock osteochondral fragments: A scoping review - Part 1.

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Abstract

[Indexed for MEDLINE] Conflict of interest statement: Declaration of Competing Interest None of the authors of this paper has a financial or personal relationship with other people or organisations that could inappropriately influence or bias the content of the paper. 18. Vet J. 2013 Apr;196(1):12-9. doi: 10.1016/j.tvjl.2012.11.009. Epub 2012 Dec 23. Canine elbow dysplasia: aetiopathogenesis and current treatment recommendations. Michelsen J(1). Author information: (1)School of Animal and Veterinary Science, Charles Sturt University, Locked Bag 588, Wagga Wagga, NSW 2678, Australia. jmichelsen@csu.edu.au Comment in Vet J. 2013 Apr;196(1):4-5. doi: 10.1016/j.tvjl.2012.12.024. Elbow dysplasia is a common debilitating condition of large and giant breed dogs. Environmental factors and a complex genetic heritability play a role in predisposing dogs to elbow dysplasia with two aetiopathogeneses suggested for the development of the disease. Osteochondrosis was initially thought to cause elbow dysplasia, but more recent evidence has strongly supported various forms of joint incongruity as the most likely cause in most cases. Radioulnar length discrepancies and humeroulnar curvature mismatch have been implicated as the cause of medial coronoid disease and ununited anconeal process, but radial incisure incongruity and biceps/brachialis muscle forces could possibly play a role in some dogs. Treatment of elbow dysplasia should address articular pathology, such as fragmented coronoid process, osteochondrosis, cartilage damage and ununited anconeal process as well as any identified underlying causes. Finally, several palliative procedures have been developed to address more advanced elbow disease and might offer improved outcomes compared to conventional medical management. Copyright © 2012 Elsevier Ltd. All rights reserved. DOI: 10.1016/j.tvjl.2012.11.009