Orthonotes
Orthonotes
by the.bonestories
v3.0 Fusion
v3.0 Fusion
trauma topic hub

Fat Embolism Syndrome

Classically 24–72 h after long‑bone/pelvic fractures or IM reaming; triad: hypoxemia, neurological signs, petechiae. Diagnosis is clinical; supported by Gurd’s criteria (1 major + 4 minor) or Schonfeld score (>5). ABG hypoxemia, CXR fluffy infiltrates; brain MRI 'starfield' pattern on DWI. Prevention: early stabilization of long bone fractures; careful reaming/venting. Management: supportive (oxygen/PEEP, fluids), avoid overload; steroids controversial.

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Classically 24–72 h after long‑bone/pelvic fractures or IM reaming; triad: hypoxemia, neurological signs, petechiae. Diagnosis is clinical; supported by Gurd’s criteria (1 major + 4 minor) or Schonfeld score (>5). ABG hypoxemia, CXR fluffy infiltrates; brain MRI 'starfield' pattern on DWI. Prevention: early stabilization of long bone fractures; careful reaming/venting. Management: supportive (oxygen/PEEP, fluids), avoid overload; steroids controversial.
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Question 1

What is the classical triad of symptoms in Fat Embolism Syndrome?

Question 2

Which of the following is a major criterion for the diagnosis of Fat Embolism Syndrome according to Gurd's criteria?

Question 3

What is the most common cause of Fat Embolism Syndrome?

Question 4

Which of the following findings is MOST characteristic of Fat Embolism Syndrome on brain MRI?

Question 5

What is the recommended management for Fat Embolism Syndrome?

Question 6

Which of the following is a minor criterion for Fat Embolism Syndrome according to Gurd's criteria?

Question 7

Fat Embolism Syndrome typically develops within how many hours after injury?

Question 8

What is the role of steroids in the management of Fat Embolism Syndrome?

Question 9

Which of the following radiological findings is associated with Fat Embolism Syndrome?

Question 10

What is the primary mechanism of fat embolism in Fat Embolism Syndrome?