Trunnionosis = corrosion/wear at head–neck modular junction of THA. Mechanism: fretting + crevice corrosion, leading to metal ion release. Clinical: unexplained pain, swelling, adverse local tissue reaction (ALTR). Diagnosis: ESR/CRP to rule out infection; elevated cobalt/chromium; MRI (MARS) for pseudotumor. Management: revision with ceramic heads, titanium sleeves; avoid further corrosion.
Which ion is predominantly released in cases of trunnion corrosion involving a cobalt-chromium head?
A patient presents with new-onset hip pain and a palpable mass months after THA with a CoCr head. Which diagnostic test is most appropriate to assess for trunnionosis?
What is a common clinical feature of adverse local tissue reaction (ALTR) due to trunnionosis?
Which of the following factors is NOT associated with an increased risk of trunnionosis?
In patients with suspected trunnionosis, what is the significance of measuring serum cobalt and chromium levels?
What adverse tissue reaction is commonly associated with elevated cobalt levels in patients with trunnionosis?
What is the recommended management for a patient diagnosed with trunnionosis?
Which of the following is NOT a distinguishing feature of trunnionosis compared to metal-on-metal (MoM) bearing wear?
What is a key characteristic of the modular taper junction in total hip arthroplasty?
What is the primary mechanism involved in trunnionosis at the head–neck junction of a total hip arthroplasty (THA)?
