Wiltse etiologic types: I dysplastic, II isthmic (IIA lytic, IIB elongated pars, IIC acute pars), III degenerative, IV traumatic (other than pars), V pathologic, VI iatrogenic. Meyerding grades I–V (25% increments) quantify slip; slip angle and pelvic incidence inform reduction strategy. Adult degenerative L4–5 listhesis: decompression with fusion when instability/foraminal stenosis present. High‑grade isthmic L5–S1 in adolescents may need reduction and circumferential fusion; monitor for L5 neuropraxia. Nonoperative measures (activity modification, core strengthening) first in low‑grade without neurologic compromise.
What is the primary aetiology of Type I spondylolisthesis according to the Wiltse classification?
In which age group is Type II isthmic spondylolisthesis most commonly observed?
Which subtype of Type II isthmic spondylolisthesis is characterized by a fatigue stress fracture of the pars interarticularis?
What is the most common level for degenerative spondylolisthesis?
Which grade of Meyerding classification indicates a slip of 51-75%?
Which type of spondylolisthesis is most likely to require surgical management?
What is the primary nonoperative management strategy for low-grade spondylolisthesis without neurologic compromise?
In the context of spondylolisthesis, what does 'spondyloptosis' refer to?
Which classification type is associated with acute fractures of the pars interarticularis?
In terms of pelvic incidence, what is its relevance in managing high-grade spondylolisthesis?
