Fat Embolism Syndrome
Classically 24–72 h after long‑bone/pelvic fractures or IM reaming; triad: hypoxemia, neurological signs, petechiae. Diagnosis is clinical; supported by Gurd’s criteria (1 major + 4 minor) or Schonfeld score (>5). ABG hypoxemia, CXR fluffy infiltrates; brain MRI 'starfield' pattern on DWI. Prevention: early stabilization of long bone fractures; careful reaming/venting. Management: supportive (oxygen/PEEP, fluids), avoid overload; steroids controversial.
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10What is the classical triad of symptoms in Fat Embolism Syndrome?
mcqWhich of the following is a major criterion for the diagnosis of Fat Embolism Syndrome according to Gurd's cri...
mcqWhat is the most common cause of Fat Embolism Syndrome?
mcqWhich of the following findings is MOST characteristic of Fat Embolism Syndrome on brain MRI?
mcqWhat is the recommended management for Fat Embolism Syndrome?
mcqWhich of the following is a minor criterion for Fat Embolism Syndrome according to Gurd's criteria?
mcqFat Embolism Syndrome typically develops within how many hours after injury?
mcqWhat is the role of steroids in the management of Fat Embolism Syndrome?
mcqWhich of the following radiological findings is associated with Fat Embolism Syndrome?
mcqWhat is the primary mechanism of fat embolism in Fat Embolism Syndrome?